> [Xylitol] is also produced by our own bodies through a process known as the glucuronate pathway, one of the pathways by which we metabolize glucose. So how did the authors ensure they were investigating the relationship between dietary xylitol intake and MACE risk? They didn’t.
> In other words, this study provides no evidence whatsoever that dietary xylitol intake has any association with MACE, yet still the authors insist on linking the two in their conclusions
> First erythritol, now xylitol. This research group followed the same flawed design and ended up with the same flawed conclusions. SSDD – same [study], different day.
Comments
From the article you linked, for the lazy:
> [Xylitol] is also produced by our own bodies through a process known as the glucuronate pathway, one of the pathways by which we metabolize glucose. So how did the authors ensure they were investigating the relationship between dietary xylitol intake and MACE risk? They didn’t.
> In other words, this study provides no evidence whatsoever that dietary xylitol intake has any association with MACE, yet still the authors insist on linking the two in their conclusions
> First erythritol, now xylitol. This research group followed the same flawed design and ended up with the same flawed conclusions. SSDD – same [study], different day.